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GPR35 agonists (YE120, zaprinast, and pamoic acid) promoted wound mend within a focus-dependent manner independently of cell proliferation, whereas a selected GPR35 antagonist CID2745687, forskolin, and pertussis toxin reversed the YE120-induced outcome. YE120 amplified the mRNA expression of fibronectin and its receptor integrin αfive, and ERK1/t

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thirteen,thirty In addition, CCL2 has become claimed to improve osteoclastogenesis by inducing the expression of receptor activator of NF-κB (RANK) by osteoclast progenitors.31 RANK ligand (RANKL) further more encourages the differentiation of osteoclast progenitors into practical osteoclasts, which could aggravate DP by resulting in alveolar bone

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